It has been revealed the fact that the covid 19 vagus nerve inflammation could be triggering dysautonomia. Check out the latest reports about this below.
New revelations about covid 19
If you’re still experiencing symptoms long after recovering from COVID-19, you’re not alone. Many people report fatigue, brain fog, and a range of other issues, from heart palpitations to gastrointestinal problems. However, new research may offer some answers about what’s causing these lingering symptoms.
A recent study published in Acta Neuropathologica suggests that the virus may damage the nerves of the autonomic nervous system, leading to an inflammatory response that can cause ongoing issues. Specifically, the researchers looked at the vagus nerves in 27 patients who had died from COVID-19 and compared them to five controls who had died of other causes.
The vagus nerve is a critical part of the ANS, which plays a role in regulating many essential functions, such as heart rate, digestion, and immune response. It also controls the body’s response to inflammation, which can lead to symptoms like fatigue, nausea, and pain. While the study’s findings are still preliminary, they offer hope for those struggling with long-term COVID symptoms.
The researchers detected SARS-CoV-2 RNA in vagus nerve samples obtained from deceased patients with severe COVID-19 showing direct infection of the nerve was accompanied by inflammatory cell infiltration composed mostly of monocytes—a type of white blood cell that finds and destroys germs and eliminates infected cells. Their analysis revealed a “strong enrichment of genes regulating antiviral responses and interferon signaling,” supporting the idea that vagus nerve inflammation is a common phenomenon with COVID-19.
It is concerning to learn that there is a direct correlation between SARS-CoV-2 viral RNA load and central nervous system dysfunction. Recent research has shown that the virus was present in the vagus nerve, and it is important to determine if this contributes to dysautonomia observed in long COVID. The study conducted on a cohort of 323 patients admitted to the emergency room between Feb. 13, 2020, and Aug. 15, 2022, categorized by whether they had mild, moderate, severe, critical, or lethal COVID-19, found that the respiratory rate increased in survivors but decreased in non-survivors of critical COVID-19. These results suggest that SARS-CoV-2 induces vagus nerve inflammation followed by autonomic dysfunction, which could contribute to critical disease courses. It is crucial to continue researching and gathering more information to help those affected by COVID-19.